Welcome, welcome! I very much enjoyed reading your post. I have also found that different oils can create a certain mood or attitude. Aroha Kyaku for sure is one of them. Makes me feel strong, purposeful, wanting to be productive. When I need to do some work on the car or around the house, it is my go-to. Especially if I will have to break a sweat. Great for going to the gym for me as well.
Other oils can make me more focused, calm, at peace. Hainan 2005, Assam 3000 come to mind.
Sometimes I pick an oil that fits the mood or situation, and sometime I pick one to change it.
Best,
Phil
Hello Alkhadra! Great post and welcome to this awesome forum. There are some great people on here. You have some great oils there and I enjoy your description of how they affect you.
For me, Kynam No1 has the most deep and profound effects. I'm sure everyone here has read it a million times from me, but I can't help it. Kynam No1 is like perfect meditation for me. Each deep breath of it helps me find that perfectly still place where the last thought has ended, but the next has yet to begin. But it goes beyond that, its truly medicinal for me, my natural anxiolytic and antidepressant.
Agree with Phil as well, sometimes I pick an oil for the mood/situation, but sometimes my mood requires changing an I pick an oil to help accommodate. Other times I let the oils tell me what to wear.
-Brian
For me, Kynam No1 has the most deep and profound effects. I'm sure everyone here has read it a million times from me, but I can't help it. Kynam No1 is like perfect meditation for me. Each deep breath of it helps me find that perfectly still place where the last thought has ended, but the next has yet to begin. But it goes beyond that, its truly medicinal for me, my natural anxiolytic and antidepressant.
Agree with Phil as well, sometimes I pick an oil for the mood/situation, but sometimes my mood requires changing an I pick an oil to help accommodate. Other times I let the oils tell me what to wear.
-Brian
A
I would definitely agree with you on the Hainan 2005 and Assam 3000. Although I've only had a small sample of Hainan, it was very calming, with a slightly sour tinge (that might be the orange-peel note everyone was talking about).
The Assam 3000 is a beauty though, it almost has a tartness that reminds me of gourmet jam.
Which oil from your collection do you think has the strongest effect on you? and what is that effect is you don't mind sharing with us!
The Assam 3000 is a beauty though, it almost has a tartness that reminds me of gourmet jam.
Which oil from your collection do you think has the strongest effect on you? and what is that effect is you don't mind sharing with us!
A
bhanny,
I've read a lot about you talking about Kynam No 1. You've always got me excited about it and yet I've always been reluctant to wear it, waiting for the right day and occasion. I have sniffed it from the vial multiple times to get an idea of what it smells like and I'd have to agree with you on it being medicinal. I'll definitely be sure to share with you my experiences once I give it a few wears.
I've read a lot about you talking about Kynam No 1. You've always got me excited about it and yet I've always been reluctant to wear it, waiting for the right day and occasion. I have sniffed it from the vial multiple times to get an idea of what it smells like and I'd have to agree with you on it being medicinal. I'll definitely be sure to share with you my experiences once I give it a few wears.
bhanny,
I've read a lot about you talking about Kynam No 1. You've always got me excited about it and yet I've always been reluctant to wear it, waiting for the right day and occasion. I have sniffed it from the vial multiple times to get an idea of what it smells like and I'd have to agree with you on it being medicinal. I'll definitely be sure to share with you my experiences once I give it a few wears.
I've read a lot about you talking about Kynam No 1. You've always got me excited about it and yet I've always been reluctant to wear it, waiting for the right day and occasion. I have sniffed it from the vial multiple times to get an idea of what it smells like and I'd have to agree with you on it being medicinal. I'll definitely be sure to share with you my experiences once I give it a few wears.
Possible active compounds
(Crossposted from Ouddict.com) http://www.ouddict.com/threads/intellectual-discourse.72/
My money is on the Eudesmol fractions of Oud.
Below is an abstract of the study of just one of the compounds contained within.
alpha-eudesmol, a P/Q-type Ca(2+) channel blocker, inhibits neurogenic vasodilation and extravasation following electrical stimulation of trigeminal ganglion.
Asakura K1, Kanemasa T, Minagawa K, Kagawa K, Yagami T, Nakajima M, Ninomiya M.
Author information
Abstract
In this study, we investigated the effect of alpha-eudesmol, which potently inhibits the presynaptic omega-agatoxin IVA-sensitive (P/Q-type) Ca(2+) channel, on neurogenic inflammation following electrical stimulation of rat trigeminal ganglion. Treatment with alpha-eudesmol (0.1-1 mg/kg. i.v.) dose-dependently attenuated neurogenic vasodilation in facial skin monitored by a laser Doppler flowmetry. In addition, alpha-eudesmol (1 mg/kg. i.v.) significantly decreased dural plasma extravasation in analysis using Evans blue as a plasma marker. On the other hand, alpha-eudesmol (1 mg/kg, i.v.) did not affect mean arterial blood pressure in rats. The calcitonin gene-related peptide (CGRP) and substance P (SP) released from activated sensory nerves have recently been suggested to be associated with the neurogenic inflammation. In this study, we also showed that alpha-eudesmol (0.45-45 microM) concentration-dependently inhibits the depolarization-evoked CGRP and SP release from sensory nerve terminals in spinal cord slices. These results indicate that the anti-neurogenic inflammation action of alpha-eudesmol, which does not affect the cardiovascular system, may be due to its presynaptic inhibition of the neuropeptide release from perivascular trigeminal terminals. We also suggest that the omega-agatoxin IVA-sensitive Ca(2+) channel blocker, alpha-eudesmol, may become useful for the treatment of the neurogenic inflammation in the trigemino-vascular system such as migraine.
(Crossposted from Ouddict.com) http://www.ouddict.com/threads/intellectual-discourse.72/
My money is on the Eudesmol fractions of Oud.
Below is an abstract of the study of just one of the compounds contained within.
alpha-eudesmol, a P/Q-type Ca(2+) channel blocker, inhibits neurogenic vasodilation and extravasation following electrical stimulation of trigeminal ganglion.
Asakura K1, Kanemasa T, Minagawa K, Kagawa K, Yagami T, Nakajima M, Ninomiya M.
Author information
Abstract
In this study, we investigated the effect of alpha-eudesmol, which potently inhibits the presynaptic omega-agatoxin IVA-sensitive (P/Q-type) Ca(2+) channel, on neurogenic inflammation following electrical stimulation of rat trigeminal ganglion. Treatment with alpha-eudesmol (0.1-1 mg/kg. i.v.) dose-dependently attenuated neurogenic vasodilation in facial skin monitored by a laser Doppler flowmetry. In addition, alpha-eudesmol (1 mg/kg. i.v.) significantly decreased dural plasma extravasation in analysis using Evans blue as a plasma marker. On the other hand, alpha-eudesmol (1 mg/kg, i.v.) did not affect mean arterial blood pressure in rats. The calcitonin gene-related peptide (CGRP) and substance P (SP) released from activated sensory nerves have recently been suggested to be associated with the neurogenic inflammation. In this study, we also showed that alpha-eudesmol (0.45-45 microM) concentration-dependently inhibits the depolarization-evoked CGRP and SP release from sensory nerve terminals in spinal cord slices. These results indicate that the anti-neurogenic inflammation action of alpha-eudesmol, which does not affect the cardiovascular system, may be due to its presynaptic inhibition of the neuropeptide release from perivascular trigeminal terminals. We also suggest that the omega-agatoxin IVA-sensitive Ca(2+) channel blocker, alpha-eudesmol, may become useful for the treatment of the neurogenic inflammation in the trigemino-vascular system such as migraine.